Descriptive, not prescriptive

The hallmarks are the field’s organizing scheme for the biology of aging. This map uses them to explain how the network is structured — which mechanism gets which site. It does not claim the network “targets aging,” and mapping a hallmark to a site is not a claim that anything slows or reverses it.

12
hallmarks (2023 set)
4
with a live site
8
no dedicated site yet
#Hallmark of agingMechanism site
1Genomic instability
Accumulating DNA damage and mutations across the nuclear and mitochondrial genome over a lifetime.
no dedicated site yet
2Telomere attrition
Progressive shortening of the protective chromosome-end caps, limiting replicative capacity.
TeloiX — live
3Epigenetic alterations
Age-associated changes in DNA methylation, histone modification and chromatin remodeling.
no dedicated site yet
4Loss of proteostasis
Declining protein quality control, chaperone function and clearance of damaged proteins.
no dedicated site yet
5Disabled macroautophagy
Reduced autophagic turnover of damaged organelles and aggregates (added as a distinct hallmark in 2023).
no dedicated site yet
6Deregulated nutrient-sensing
Dysregulation of the insulin/IGF-1, mTOR, AMPK and sirtuin nutrient-sensing network.
MtoriX — live
7Mitochondrial dysfunction
Declining mitochondrial efficiency, altered dynamics and increased reactive-oxygen-species leakage.
MitoiX — live
8Cellular senescence
Accumulation of growth-arrested cells with a pro-inflammatory secretory phenotype (SASP).
SenesiQ — live
9Stem cell exhaustion
Decline in the regenerative capacity of tissue-resident stem-cell pools.
no dedicated site yet
10Altered intercellular communication
Disrupted endocrine, neuronal and immune signaling between cells and tissues.
no dedicated site yet
11Chronic inflammation
Persistent, low-grade sterile inflammation ('inflammaging') that rises with age.
no dedicated site yet
12Dysbiosis
Age-associated shifts in the composition and function of the gut and other microbiota.
no dedicated site yet
No mapping is invented. Only four hallmarks — nutrient-sensing, cellular senescence, mitochondrial dysfunction and telomere attrition — currently have a live, source-verified mechanism site. The other eight are marked honestly as having no dedicated site yet; the hub does not assign them to planned domains that hold no content.

Source

Lopez-Otin C, Blasco MA, Partridge L, Serrano M, Kroemer G. “Hallmarks of aging: An expanding universe.” Cell 186:243-278 (2023). DOI 10.1016/j.cell.2022.11.001.

DOI verification: verified_crossref (2026-07-13). Crossref returns DOI 10.1016/j.cell.2022.11.001 = 'Hallmarks of aging: An expanding universe', Cell (Elsevier BV), volume 186, pages 243-278. Title, container, volume and page range confirmed against the primary Crossref record.